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  • 产品名称：TumorNecrosisFactorReceptorSuperfamily,Member6b,Decoy(TNFRSF6B)(N-Term)Peptide

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  • 产品厂商：PromoCell
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简单介绍：
TumorNecrosisFactorReceptorSuperfamily,Member6b,Decoy(TNFRSF6B)(N-Term)Peptide
详情介绍：
Blocked Antibody anti-DcR3 (Decoy Receptor For FASL and LIGHT) (N-Term) antibody (ABIN1002198)
Characteristics 16 amino acids near the amino terminus of human DcR3.
Application Notes DcR3 peptide is used for blocking the activity of Anti-DcR3 . Incubating the peptide with equal volume of antibody for 30 min at 37ºC usually completely blocks the antibody activity in Western blotting.
Restrictions For Research Use only
Format Liquid
Concentration 200 µg/mL
Buffer Supplied in PBS pH 7.2 (10 mM NaH₂PO₄, 10 mM Na₂HPO₄, 130 mM NaCl) containing 0.1% bovine serum albumin and 0.02% sodium azide.
Preservative Sodium azide
Precaution of Use This product contains sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.
Handling Advice For products with volumes of 200 μl or less, we recommend gently tapping the vial on a hard surface or briefly centrifuging the vial in a tabletop centrifuge to dislodge any liquid in the container’s cap. The peptide solution should be gently mixed before use.
Storage -20 °C
Storage Comment Store at -20°C, stable for one year.
Expiry Date 12 months
Product cited in: Kawai, Adachi, Ogawa, Takeda, Akira: "Unresponsiveness of MyD88-deficient mice to endotoxin." in: Immunity, Vol. 11, Issue 1, pp. 115-22, 1999 (PubMed).

Medzhitov, Preston-Hurlburt, Kopp, Stadlen, Chen, Ghosh, Janeway: "MyD88 is an adaptor protein in the hToll/IL-1 receptor family signaling pathways." in: Molecular cell, Vol. 2, Issue 2, pp. 253-8, 1998 (PubMed).

Background publications Adachi, Kawai, Takeda, Matsumoto, Tsutsui, Sakagami, Nakanishi, Akira: "Targeted disruption of the MyD88 gene results in loss of IL-1- and IL-18-mediated function." in: Immunity, Vol. 9, Issue 1, pp. 143-50, 1998 (PubMed).

Muzio, Ni, Feng, Dixit: "IRAK (Pelle) family member IRAK-2 and MyD88 as proximal mediators of IL-1 signaling." in: Science (New York, N.Y.), Vol. 278, Issue 5343, pp. 1612-5, 1997 (PubMed).